My delayed cytotoxic reaction on LIT

Delayed cytoxic reaction and delayed hypersentivity reaction is the same.  It takes 2 to 3 days to develop. This explains why my LIT sites started to reduce its swelling and rebounded back after the third day.

This reminds me of my tetanus skin test. I did not react to the skin test within 15 minutes. My rashes developed few hours after the intramuscular injection. What is delayed type hypersensitivity mean on my previous tetanus toxoid skin test? I never had tetanus antibody, therefore no antibody to react with the tetanus toxoid yet? Or I have other immunologic factor?  I will ask again this issue to my immunologist.

I remembered he mentioned about th1/th2 balance. My Th1 is high. Hmmm... Haha next time I propose pro-histamine to induce th2 pathway. high th1 + high th2 = balanced th1/th2 ^.^ What if my wicked proposal is valid? Yeah, I dont have to worry about tnf-alpha... Tnf-alpha will be counted by il-10. Haha I can proceed with IVF,  no need for another round of LIT ^.^  just kidding...

Poor me, my obgyne asked how many sessions of LIT do I need? I replied I don't know. If I heard it right, my immunologist said in the past, I need four sessions of LIT. But my last consultation, he said he needs to evaluate again after my next LIT. Delayed cytotoxic reaction involves th1- it is problematic for him.

https://www.boundless.com/microbiology/immunology-applications/immunity-disorders-hypersensitivity/type-iv-delayed-cell-mediated-reactions/
"Type IV hypersensitivity is often called delayed type hypersensitivity as the reaction takes two to three days to develop. Unlike the other types, it is not antibody mediated but rather is a type of cell-mediated response. CD4+ helper T cells recognize antigen in a complex with Class 2 major histocompatibility complex. The antigen-presenting cells in this case are macrophages that secrete IL-12, which stimulates the proliferation of further CD4+ Th1 cells. CD4+ T cells secrete IL-2 and interferon gamma, further inducing the release of other Th1 cytokines, thus mediating the immune response. Activated CD8+ T cells destroy target cells on contact, whereas activated macrophages produce hydrolytic enzymes and, on presentation with certain intracellular pathogens, transform into multinucleated giant cells."